Reduce your Alzheimer’s risk by 50% by treating this disease...

March 05, 2012
Volume 3    |   Issue 15

For many years, the focus on Alzheimer’s prevention has been the brain. Makes sense, right? After all, you would think that fixing problems in the brain would stop Alzheimer’s before it happens. But new research suggests that treating another disease, rather than just focusing on Alzheimer’s itself, could be an effective way to reduce your risk. In fact, a new study says you can cut your risk of Alzheimer’s in half by treating a completely different disease.

That disease is diabetes. And the study comes from Japan. In this study, researchers followed over 1,000 men and women over the age of 60. They found that those with diabetes (especially type-2 diabetes) were twice as likely to develop Alzheimer’s. But that’s not all. Diabetics have a 75% higher risk of getting other forms of dementia.

While the researchers continue to look for why this connection exists, my friend and colleague Dr. Robert Rowen has already revealed the connection. In his newsletter, Second Opinion, Dr. Rowen has showed that Alzheimer’s is really a type of diabetes. In fact, he says it’s type-3 diabetes. Here’s the evidence:

“As you may know, insulin resistance is the first step in developing type-2 diabetes. The main cause for insulin resistance is a high-fat diet. A dietary guru taught me years ago that fat tends to block insulin activity at the cell membrane, causing insulin resistance. Researchers at Mount Sinai School of Medicine have linked this fact with a novel insulin metabolic effect in the brain.

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“These researchers injected tiny amounts of insulin into the brains of rats. Then they measured the effect on glucose and lipid metabolism in the whole body. They found that the brain insulin suppresses lipolysis. This is the process that breaks down fat in triglycerides and releases fatty acids.... When you impair brain insulin function, your brain can’t suppress lipolysis. The breakdown of fats continues to add fuel to the fire of insulin resistance in a worsening spiral....”

In other words, “high blood sugar or insulin resistance might impair your neurons' ability to discard waste or handle the beta amyloid that accumulates and ultimately kills the cells.” And this leads to memory loss and Alzheimer’s.

So if you want to prevent Alzheimer’s and any form of dementia, start by treating diabetes. You can do this by eating a low-fat, low-sugar, low-carb diet. Dr. Rowen has a 100% cure rate for those who follow his protocol (which you can read all about by subscribing to his newsletter. There are also a host of spices you can add to your diet that will help. These include curry (curcumin), basil, oregano, and cumin. Studies have shown that they can help your memory and overall brain function.

But don’t stop there. Supplements can really help with diabetes and Alzheimer’s. Herbs and nutrients, like those in Metabolic Defense, can help reduce your sugar cravings and balance your blood sugar.

By treating diabetes and keeping your brain functioning at optimal levels, you can significantly reduce your risk of ever developing Alzheimer’s. And don’t miss my next alert on Saturday, as I’ll show you an interesting connection between Alzheimer’s and pain.

Your insider for better health,

Steve Kroening

Steve Kroening is the editor of Nutrient Insider, a twice-a-week email newsletter that brings you the latest healing breakthroughs from the world of nutrition and dietary supplements. For over 20 years, Steve has worked hand-in-hand with some of the nation's top doctors, including Drs. Robert Rowen, Frank Shallenberger, Nan Fuchs, William Campbell Douglass, and best-selling author James Balch. Steve is the author of the book Practical Guide to Home Remedies. As a health journalist, Steve's articles have appeared in countless magazines, blogs, and websites.


Mount Sinai website, February 16, 2011.
Neurology. 2011 Sep 20;77(12):1126-34.
Diabet Med. 2011 Oct 4. doi: 10.1111/j.1464-5491.2011.03464.x. [Epub ahead of print].
Second Opinion, September 2011.

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